NF-κB induces cytokines, adhesion molecules and angiogenic factors to be expressed at the tumor site, increasing survival and proliferation of neoplastic cells via regulation of genes related to cell-cycle control (e.g., cyclin-D1, c-MYC) and apoptosis (e.g., BCL-2, cFLIP) [74]. Here, NFKB1 is linked to neoplasm.