Knowing that the PP2A complex positively regulates several pro-inflammatory pathways, including NF-kappa B, p38 MAPK and JNK [18,27,28], and is the main target of Mid1 in epithelial lung adenocarcinoma, bronchial epithelium in asthma and primary cerebellar granule neurons [18,29,30], it was of great interest to study levels of PP2Ac in endothelial cells. This evidence concerns the gene MID1 and asthma.