The overexpression of FOXM1 in cancers has been linked to oncogenic transformation, tumour initiation, growth, progression, migration, invasion, and metastasis via impact on epithelial-mesenchymal transition, angiogenesis, the recruitment of tumour-associated macrophages, the prevention of premature cellular senescence, and chemotherapeutic drug resistance [163]. This evidence concerns the gene FOXM1 and neoplasm.