MALAT1 and posterior cortical atrophy: Since we previously reported that the overexpression of miR-423-5p was able to inhibit MALAT1-mediated proliferation, migration, and invasion of PCa cells, we evaluated the clinical significance of miR-423-5p in the TCGA-PRAD dataset using the ENCORI Pan-Cancer analysis platform and no significant correlations between the expression level of miR-423-5p and prognosis in PRAD groups was detected [16].