ERBB4 and Anxiety: Since deficits in working memory and anxiety-like behaviors have been previously linked to deficits in the ErbB4 in PV+ INs [37], thus, we postulate that the acute and late ErbB4 deficits in the hippocampus after neonatal HI may be the mechanism leading to persistent deficits in PV+ INs in the dorsal hippocampus after and contributing in part to the impaired memory and mechanisms of synaptic plasticity after HI.