Furthermore, the overexpression of FOXM1 leads to the acquisition of an EMT phenotype by activating the mesenchymal cell markers ZEB1, ZEB2, Snail2, E-cadherin, and vimentin, which are associated with increased spheroid-forming capacity and cancer stem cell surface markers (CD44 and EpCAM) [26,27]. This evidence concerns the gene EPCAM and cancer.