H19 and Stroke: Of note, the expression of H19 inversely correlated with that of miR-18a, in line with previous demonstrations that miRNAs belonging to the miR-17-92 cluster are inhibited by this lncRNA [42], and directly correlated with VEGF, a potential direct target of miR-18a [43] associated with angiogenesis and BBB breakdown, suggesting the existence of a H19/miR-18a/VEGF axis that regulates BBB permeability after stroke.