KLRD1 and endometriosis: The studies explored in this review [5,6,7,8,9,10,11,12,13,14,15,16,17,18,19,20,24] corroborate this notion, detailing a pathological local environment of endometriosis whereby the aberrant expression/activity of NK cell receptors results in less cytotoxicity (via the increased inhibition, decreased activation, and apoptosis of NK cells themselves) and contribute to a pro-inflammatory state which promotes proliferation and angiogenesis.