SOD1 and amyotrophic lateral sclerosis: Therefore, experimental processes capable of inhibiting stress mechanisms (e.g., oxidative stress, glutamate excitotoxicity; apoptosis and other related pathological processes), proteinopathy (including misfolding, aggregation and toxicity of SOD1 protein, or other misfolding proteins known to be associated with ALS, as well as related processes including autophagy or endoplasmic reticulum stress), immune mechanisms (including inflammation, reactive astrogliosis and microgliosis, leukocyte activation and so on) or processes regulating cell fate were classified in the corresponding category.