miR-200a-5p inhibits the stress-associated selenoproteins (Sel), including selenoprotein 15 (Sep15), selenoprotein t (Selt), and selenoprotein p1 (Sepp1), impairs the Sel-mediated glucose metabolism, increases the glucose uptake in cardiomyocytes and leads to cardiac hypertrophy [72]. This evidence concerns the gene SELENOT and cardiac hypertrophy.