On the other hand, both HFD and SAC groups had hepatic steatosis (Figure 1R,S, Supplementary Figure S1G), high plasma alanine transaminase (ALT) activity (Supplementary Figure S1H), and low plasma β-hydroxybutyrate (β-HB), a proxy for liver β-oxidation (Figure 1T). This evidence concerns the gene GPT and Hepatic steatosis.