Previously, we demonstrated a frequency of 36.6% of Bcl-6 translocation in GBM IDH1 mutated patients in association with highly expression of Bcl-6 at protein and messenger levels, demonstrating that Bcl-6 plays a role in the repression of apoptosis of cancer cells and its expression correlated with p53 [31]. Here, BCL6 is linked to glioblastoma.