Interestingly, based on the observed upregulation of known hypoxia target genes, such as GLUT1 and VEGFA [52,53], we have indicated that the virus infection triggers hypoxia-like conditions in Vero E6 and A549 cells upon SARS-CoV-2 infection [19], and have suggested hypoxia signaling as a mechanism of DDC and dACE2 co-regulation during infection. Here, SLC2A1 is linked to infection.