IFNG and infection: Correspondingly, Hoarau and colleagues [43] evaluated a large set of immunological and biochemical lab parameteres from 32 hospitalized patients with confirmed CHIKF, in whom they observed a robust cellular and molecular innate immune response during acute phase in all patients, characterized by activaction of NK cells, plasmocytoid dendritic cells and T cell subsets, as well as higher serum levels of IFNγ and IL-12 when CHIKF patients were compared to those with other infections and to healthy controls.