Lechien et al. [9] stated that “the pathophysiology of oropharyngeal dysphagia (OD) in COVID-19 patients is assumed to be related to the interaction of the virus with angiotensin-converting enzyme 2 (ACE2) and transmembrane protease serine 2 (TMPRSS2), proteins that are present in relevant anatomic regions for swallowing function, such as oral, pharyngeal and nasal human mucosa”. This evidence concerns the gene ACE2 and osteochondritis dissecans.