NFKB1 and essential thrombocythemia: The proteomic analysis showed that 20 proteins were altered in ET with the CALRdel52 mutation: those involved in cell signalling (Erk1/2, PTEN, Raf‐B, Rap1, Axin, ERβ, TGF‐β), cell cycle (Cdc42, Cdc2, CyclinD1, p27), apoptosis (Bcl‐Xl, c‐IAP2, NFκB p50, cPKCα, Survivin), transcription factor (eIF4B, SRC‐1), adhesion (E‐cadherin) and DNA repair (TDP1).17