CD68 and myocardial infarction: Since EEEG attenuated the infiltration of CD45+ leukocytes and CD68+ macrophages and reduced the production of inflammatory mediators in the ischemic myocardium in vivo (Figures 3A–C), it is reasoned that EEEG protects against MI-induced cardiac inflammation through a mechanism involving attenuation of endothelial pro-inflammatory activation.