A study which aimed to describe the interaction between MALAT1 and miR-125b in bladder cancer cells and their roles in cancer development found that MALAT1 directly targeted miR-125b and antagonized miR-125b expression, resulting in inhibition of apoptosis and promotion of invasion of bladder cancer cells by upregulation of the expression levels of the anti-apoptotic genes B-cell leukemia 2 (Bcl-2) and matrix metallopeptidase 13 (MMP13) [40]. The gene discussed is MALAT1; the disease is cancer.