Feng et al. [56] reported that SNHG14 enhances cell cycle progression, colony formation, invasion, migration, and proliferation, and that it inhibits apoptosis of bladder cancer cells by targeting miR-211–3p to upregulate the expression of endothelial cell-specific molecule 1 (ESM1), a soluble dermatan sulfate proteoglycan that participates in cancer progression and metastasis [57]. This evidence concerns the gene ESM1 and urinary bladder cancer.