Despite comparable expression of AICD mediators in T‐cells (FasL, TNF, and TRAIL) from KRAS mutant versus wild‐type tumors (Figure 4F–H), markedly increased apoptosis was observed in the tumor‐specific CTLs from KRAS mutant tumor tissues after cells were cocultured with autologous primary tumor cells (Figure 4I,J). The gene discussed is KRAS; the disease is neoplasm.