However, as we reported that the patient had no previous HBV infection and the AFP was only mildly elevated in the preoperative examination, we believe that the cause of the patient’s HCC may be the compression of the surrounding liver tissues by hepatic cysticercosis, which then caused inflammatory changes around the liver and eventually cirrhosis, leading to an increased risk of developing HCC, which is consistent with the idea of Kübeck M.[8] However, there is still lack of strong evidence to prove this conjecture. The gene discussed is AFP; the disease is Cirrhosis.