KLRC2 and cytomegalovirus infection: This focus is supported by HCMV biology—HCMV infection induces a striking reorganization of the NK cell compartment, with NKG2C+, KIR+ NK cells proliferating following initial infection or HCMV reactivation, leaving stable imprints on the NK cell repertoire that can last over two years (Béziat et al. 2013; Gumá et al. 2004, 2006; Rölle et al. 2014).