Ang‐II infusion causes systemic inflammation in addition to blood pressure increase, elastase perfusion damages aortic wall elastica, and peri‐aortic CaCl2 injury promotes in situ matrix remodeling, SMC apoptosis, and inflammation.[35] To test further a protective role for ILC2 in AAA development, we produced Apoe−/−Rorafl/flIl7rCre/+ and Apoe−/−Il7rCre/+ control mice and performed Ang‐II subcutaneous perfusion. Here, AGT is linked to triple-A syndrome.