A recent study reported in a mouse model of colonic adenocarcinoma that T‐cell subset accumulation (defined by their expression of the master control genes that regulate their development and function) in the tumor tissue did not differ between Rorafl/flIl7rCre/+ and Il7rCre/+ mice, including Th1 cells (Tbet‐positive), Th2 cells (GATA3‐positive), and Treg (Foxp‐positive). This evidence concerns the gene GATA3 and colon adenocarcinoma.