Besides these morphological changes, we found that ILC2 deficiency in Rorafl/flIl7rCre/+ mice led to reduced CD11b+Siglec‐F+ EOS in AAA lesions (Figure 2J), and increased lesional CD11b+Ly6G+ neutrophils, CD11c+MHC‐II+ DCs, and CD11b+Ly6Chi monocytes (Figure 2K–M). Here, ITGAM is linked to triple-A syndrome.