Also, this study did not specify the roles of different ILC2 subtypes, such as IL33‐induced ILC2 (natural ILC2), IL25‐induced ILC2 (inflammatory ILC2), Thy‐1+Sca‐1+IL‐18R+ST2−C‐Kit− ILC2, and CD103+ ILC2.[50] Although it would be ideal to isolate ILC2 from AAA lesions for direct adoptive transfer, this effort is technically impossible due to the low frequency of ILC2 in AAA lesions. This evidence concerns the gene IL18R1 and triple-A syndrome.