ITGAE and triple-A syndrome: Also, this study did not specify the roles of different ILC2 subtypes, such as IL33‐induced ILC2 (natural ILC2), IL25‐induced ILC2 (inflammatory ILC2), Thy‐1+Sca‐1+IL‐18R+ST2−C‐Kit− ILC2, and CD103+ ILC2.[50] Although it would be ideal to isolate ILC2 from AAA lesions for direct adoptive transfer, this effort is technically impossible due to the low frequency of ILC2 in AAA lesions.