After adjusting for potential confounders based on prior knowledge (Model 1 adjusted for sex, age, BMI, arterial hypertension, diabetes mellitus, and FBG; Model 2 further adjusted for ALT, AST, IgG, cirrhosis, fibrosis of biopsy, interface hepatitis, biochemical response, and corticosteroid dose) by a multivariable regression model, hepatic steatosis was found to independently predict biochemical response (P < 0.001 of all models) (Table 3). This evidence concerns the gene GPT and fatty liver disease.