In addition, further multivariate-adjusted analysis identified hepatic steatosis as an independent predictor of unsatisfactory outcomes in two models (Model 1 adjusted for sex, age, BMI, arterial hypertension, diabetes mellitus, and FBG; Model 2 further adjusted for ALT, AST, IgG, cirrhosis, fibrosis of biopsy, interface hepatitis, biochemical response, and corticosteroid dose) (Table 4). The gene discussed is GPT; the disease is fibrosis.