Studies have shown that the combination of ST1326 and ABT199 (Bcl-2 inhibitor) can enhance the anti-acute myeloid leukemia (AML) effect of the latter, indicating that cpt1a may become a potential drug target for the treatment of AML (Ricciardi et al., 2015; Mao et al., 2021). This evidence concerns the gene BCL2 and acute myeloid leukemia.