Given CD20+ B cells and CD38+ T cells are involved in activating cytotoxic T cells for killing tumour cells, such distributed pattern seemed to show that FOXP3+ Tregs and CD66b+ neutrophils populations may synergistically attenuate this process (average length of CD38‐FOXP3 connections and number of CD20–CD20 connections, r2 = 0.39; number of CD38‐CD66b connections and number of CD20‐CD20 connections, r2 = −0.31). The gene discussed is MS4A1; the disease is neoplasm.