RSPH3 and situs inversus: Normal visceral placement is driven by the dynein of nodal cilia, so patients with gene mutations encoding non-dynein structural components such as RSPH1 (Knowles et al., 2014), RSPH3 (Jeanson et al., 2015), and HYDIN (Olbrich et al., 2012) have not been reported for situs inversus.