A previous study documented that in patients acutely admitted with asthma, elevated suPAR concentrations together with blood eosinophil count < 150 cells/μL at the time of hospital admission were associated with both 365-day all-cause readmission and mortality, implying that in asthma, the uPAR pathway associates with non-T2 asthma but is implicated in neutrophils and T1/T17 T-cells that are thought to be part of the pathogenesis of the non-T2 asthma endotypes [14,16,17]. The gene discussed is PLAUR; the disease is asthma.