In most severe COVID-19 cases, an overwhelming immune response leads to an increased level of cytokines; such as tumor necrosis factor-α (TNF-α), interleukin-1(IL-1), interleukin-6 (IL-6), (interleukin-7) IL-7, interleukin-8 (IL-8), interleukin-17 (IL-17), which are supposed to be responsible for acute respiratory distress syndrome (ADRS), instead of viral activity [3]. This evidence concerns the gene CXCL8 and COVID-19.