RAG2 and stroke disorder: We determined in all groups, except of NSG mice, an acute deficit in the rotarod performance indicated by a ≈ 40% shorter time spent on the rod acutely after stroke (3dpi to bl: WT: −48%, p = 0.020, WT-Tacr: −35%, p = 0.048; Rag2-/- = −43%, p = 0.037; NSG = −22%, p = 0.549) with a gradual recovery (Figure 3B).