As shown in Figures 4A–D, patients with the CMTM7-high phenotype had remarkably higher enrichment scores of chemokines, paired receptors, MHC molecules, and immunomodulators (Figures 4A–D), which were involved in recruiting effector tumor-inflamed immune cells, such as CD8+ T cells, macrophages, and antigen-presenting cells. This evidence concerns the gene HLA-C and neoplasm.