Likewise, it has previously been shown that tumor‐derived CCL2 trigger effector T cell chemotaxis both in vitro and in vivo,[35] and CCL3 and CCL4 can also induce the recruitment of effector T cells.[36] We suspect that 6J1 might activate Rabs (e.g., Rab5 or Rab27), to induce the secretion of chemokines or cytokines. This evidence concerns the gene CCL2 and neoplasm.