Furthermore, even though the present findings were similar to the attenuated hypertensive effects we reported in OVLT lesioned rats during “AngII‐salt induced hypertension” (Collister et al., 2013), OVLT fibers of passage through the MnPO remained intact in the current study and could therefore compensate for the remaining hypertensive effects of the synergistic AngII‐high salt signal. This evidence concerns the gene AGT and hypertensive disorder.