Further, we investigated the effects of THGP on exogenously applied sulfide-induced paw allodynia, which is dependent on Cav3.2 [17,18,21,23], and on visceral pain accompanying cystitis and pancreatitis, known to involve the endogenous sulfide/Cav3.2 pathway [19,[29], [30], [31], [32]], in mice. This evidence concerns the gene CACNA1H and pancreatitis.