SARS-CoV-2 may contribute to Ang II-induced thrombosis by an imbalance in ACE2 levels, causing angiotensin-induced coagulopathy, with a high risk of thrombosis, comprising platelet activation, thrombin generation, and endothelial damage via the type 1 angiotensin II receptor [54]. This evidence concerns the gene AGT and deep vein thrombosis.