AXL and Zika virus infectious disease: Recent studies demonstrated that DCs are permissive to ZIKV infection through several adhesion factors such as DC-SIGN, AXL, TYRO3 [80,81,82,83], and could mediate an antagonistic response to type I interferon [84] and thus, they may play an important role in the pathogenesis of the disease and vertical transmission through their location in placental tissues.