Since COVID-19 dysregulates lipoproteins profiles in a way that increases the atherosclerotic risk (i.e., elevated ApoB/Apo100, reduced SPC signal, ...) [21] such long recovery times raise the possibility that the serum-altered lipoprotein composition may inflict significant damage in the arteries, engrossing the atheroma plaque. This evidence concerns the gene APOB and COVID-19.