It was demonstrated that an increased expression of miR-20a could promote the activation of the NFκB pathway by targeting NFKBIB (alternative name IκBβ) and result in the increased expression of p65, livin and survivin, which potentially contribute to a decrease in the gastric cancer cell apoptosis induced by cisplatin and chemoresistance [96]. This evidence concerns the gene NFKBIB and gastric cancer.