Moreover, high serum and tissue levels of CXCL10 have been observed in organ-specific autoimmune pathologies such as type 1 diabetes mellitus, Graves’ disease, or other autoimmune thyroiditis, and Graves’ ophthalmopathy, as well as in systemic autoimmune pathologies such as rheumatoid arthritis, systemic lupus erythematosus, systemic sclerosis, and cryoglobulinaemia in the context of hepatitis C [2]. This evidence concerns the gene CXCL10 and hepatitis C virus infection.