GAL and neoplasm: Our laboratory has demonstrated that tumor growth rates are disrupted when Gal-1-binding glycan ligands on anti-tumor T cells are inhibited by a fluorinated analog of N-acetylglucosamine, 2-acetamido-1,3,6-tri-O-acetyl-4-deoxy-4-fluoro-D-glucopyranose (4-F-GlcNAc) [144].