Gong et al. [5] indicated that, in addition to upregulated expression of BIM by EGFR-TKI in apoptosis induction, the effector pro-apoptotic protein BAX displays altered localization from the nucleus to the cytoplasm (likely the mitochondria) in erlotinib-treated apoptotic H3255 lung cancer cells. Here, EGFR is linked to lung carcinoma.