Friedreich’s ataxia (FRDA), the most common inherited ataxia [1], is a rare autosomal recessive neurodegenerative disorder due to the homozygous pathological expansion of guanine-adenine-adenine (GAA) triplet repeats, commonly in the range of 70 to 1700, in the first intron of the FXN gene located on chromosome 9q13-q21.1, which encodes for the mitochondrial protein frataxin [2]. This evidence concerns the gene FXN and Friedreich ataxia.