Higher frequency of the inactivating NQO1 SNP (C609T), which functions as a highly inducible phase II antioxidant enzyme that catalyzes quinones to hydroxyquinones, was observed in infants diagnosed with BPD, and all infants with a birth weight of less than 1000 g and at least one copy of the SNP later developed BPD [60]. The gene discussed is NQO1; the disease is bronchopulmonary dysplasia.