LGALS9 and gastric neoplasm: The interaction between EP11-CLDN7 and CD8+ T cells was dominated by the interactions of LGALS9—HAVCR2 and HVEM—CD160 both of which are well-known checkpoints that inhibit CD8 + T cell functionality (Additional file 1: Fig. S10g), suggesting an immunosuppressive function of the gastric tumor cells.