ACE2 and Genetic thrombotic microangiopathy: However, endothelial damage in the blood–neuron interface, retrograde axonal transmission, infected leukocyte‐mediated transportation (Trojan horse), cytokine storming, immune complex‐mediated reactions, angiotensin‐converting enzyme‐2 (ACE‐2) mediated virus invasion, and thrombotic microangiopathy are the most accepted theories.13, 14, 15