SOAT1 and neoplasm: The results showed that NLRP1 and NLRP3 were both involved in some tumor immune-related signaling pathways, including pathways of chemokines, leukocyte transendothelial migration, cell adhesion molecules (CAMs), cytokine-cytokine receptor interaction, mitogen-activated protein kinase (MAPK), Janus kinase (JAK)/signal transducer and activator of transcription (STAT) (Table 4, Figure 8A and 8B), suggesting NLRP1/NLRP3 may participate in the same tumor immune-related pathways to regulate tumor immune cell infiltration.