Thus, these data further support the idea that HLA-EUL40 CD8T cells stay at the frontier between adaptive, memory NK, unconventional (γδT) and conventional (αβT) T cell responses and concur together with CD57+/NKG2C+ NK and δ2-γδT to the effector cell arsenal mobilized to cure infection (Figure 7). The gene discussed is KLRC2; the disease is infection.