NFKB1 and infection: However, MHV-68 infection can sustainably induce transcription of SINE ncRNA, which is explained by Xiaonan Dong et al.: Inducing phosphorylation degradation of the RelA/p65 subunit of NF-κB in the pre-MHV-68 infection period to blunt the NF-κB transcription response, it is associated with IKKβ kinase (42).