CAMK2G and catecholaminergic polymorphic ventricular tachycardia: They showed that a selective CaMKII autocamtide-2-related inhibitory peptide transducing with adeno-associated virus (AAV) can correct BTHH and CPVT Ca2+ transient amplitude and reduce diastolic Ca2+ concentrations to a base level, resulting in the suppression of arrhythmias in murine models.