suggested that in asthma, ILCs and other cells establish a mutually balanced gene repression program (repressors Nr4a2, Zeb1, Bach2 and JunD) and preparation programs (signaling activators Fhl2, FosB, Stat6, Srebf2 and MPP7) in response to repeated stimulation of allergens. The gene discussed is JUND; the disease is asthma.