CD8A and viral infectious disease: Fabp4 is characterized to mediate lipid handling and metabolism and has been proven to upregulate CD8+ tissue-resident memory T cells of alveolar macrophage (MФ) after acute viral infection (18), and our real-time PCR data also showed that CoV2+ hamsters that received IFN-λ inoculation exhibited more induction of Fabp4 at 3 and 7 dpi (Figure 6C).